“showed a trend”

Patient with SC collected showed a trend toward significantly more ventilator-free days (6.5 vs. 0, P = 0.053).

Assessment of the regulatory CD25+FoxP3+ population also showed a trend for an increased frequency in the CD4+ subset ( p = 0.053) ( Figure 3C ) and an increased frequency for the CD4-CD8- subset ( p = 0.036) ( Figure 3D ) for patients ( n = 16, P5-20) compared to controls ( n = 20).

A haplotype of all four investigated SNPs (rs3212227-rs17860508-rs10045431-rs6887695) showed a trend for association with CD (p = 0.053); however, this potential association signal did not reach statistical significance and was uncorrected for multiple testing.

Lambda (the path length normalised for network size and connectivity), as a measure of global network integration, showed a trend for a difference in trajectories over the 24-week study period in the beta band (MMRM, F(2,279) = 2.97, p = 0.053; ICC = 0.083) (See Table 4 and Figure 5 ).

Transplants using AB donor hearts showed a trend toward significantly increased hourly IT risk with IT ≥ 3 h at 90 days (HR = 1.46, p = .053) but did not show significant risk elevation at other IT or follow‐up strata ( p > .050, Table 4 ).

Tumor grade was significantly negatively correlated with the expression of KRT5 (rs = −0.225; p = 0.029) and showed a trend for a negative correlation with AR (rs = −0.201; p = 0.053).

In the Cox regression analysis, CILP-1 levels showed a trend as a risk factor for all-cause mortality [crude HR for an increase of 500 pg/mL in CILP-1: 1.03 (95%CI: 1.00–1.06), p = 0.053].

The cytokine‐linked hematopoietic signaling in cancer pathways profile showed a trend toward lower scores in TgAD rats relative to WT (F (1,24) = 4.136, p = 0.053, η p 2=0.147), with no sex effect or interaction (Fs (1,24) ≤ 0.129, ps ≥ 0.723; Fig. 1 D).

Patients younger than 60 years showed a trend towards higher likelihood of BRAF mutations (OR = 1.88, 95% CI: 0.99–3.70, p = 0.053).

The repeated measures ANOVA for HRF showed a trend for the main effect of Time, F (1, 52) = 3.91, p = 0.053, η p a r t i a l 2 = 0.07, indicating an increase in heart rate after stress in both groups.

Heart rate changes showed a trend ( p = 0.053) but did not reach statistical significance between groups ( Table 5 ). 3.4.

However, the cognitive defect level, and scores of SA showed a trend of correlation ( r = .137, P = .053).

In this context, although not statistically significant, weight showed a trend ( p = 0.053) towards significance, probably due to the large representation of male subjects both in the recurrent group and in the whole series.

Multivariable Cox Proportional Hazards Regression In multivariable Cox proportional hazards regression analysis adjusting for age, sex, and level of obstruction, secretory obstruction (vs structural obstruction) showed a trend toward lower failure risk in the secretory group compared with the structural group (HR, 0.28; 95% CI, 0.077–1.015; p = 0.053) ( Table 3 ; Supplementary Figure S1 ). 4.

BDNF, again, showed a trend for being positively associated with training intensity [hr/week] ( ρ = 0.263, p = .053).

The comparison between enchondroma and chondrosarcoma grade 1/ACT for miR-143-3p showed a trend toward lower expression in chondrosarcoma G1/ACT (p = 0.053).

Unadjusted analyses using the median cut point showed that the low HIF1α expression was significantly associated with better LRC [HR (95% CI) = 0.58 (0.38–0.89), P = 0.011] as well as OS [HR (95% CI) = 0.62 (0.42–0.91), P = 0.016], and showed a trend towards improved PFS [HR (95% CI) = 0.69 (0.47–1.01), P = 0.053, Fig. 2a–c ].

As a post hoc analysis, connectivity of the right VTA\SNc to a (3 mm) spherical ROI centered on the NAcc peak coordinate showed a trend level positive correlation with BAS drive scores (r = 0.41, p = 0.053, partial correlation controlling for age and PDS score).

An analysis in the subgroup without diabetes showed a trend toward an improvement in the primary endpoint in the morphine group (22.5% [14–33] vs. 27.5% [22–41], p = 0.053).

Importantly, while the improvement in LDL-C and HDL-C did not differ significantly between genotype groups—suggesting that the beneficial effects of aquatic HIIT on these lipids are largely independent of ACE I/D polymorphism—post-intervention lipid profiles revealed genotype-dependent patterns: adjusted mean TG levels were significantly lower in the ID/DD group compared to the II group ( p = 0.000), and HDL-C levels showed a trend toward being higher in the ID/DD group ( p = 0.053).

Follow-up showed a trend toward fewer complications (2.5% vs. 8.5%, p = 0.053).

The HIIT showed a trend to reduce apoE ( p = 0.053) and reduced CETP proteins ( p < 0.01) in T2DM participants ( Figure 1C ).

The MoCA showed a trend (p = 0.053), not reaching statistical significance ( Table 6 ).

Medium and small body size were not significantly associated with NT after adjustment, although medium-sized dogs again showed a trend (OR = 1.42, 95% CI: 0.99–2.05, p = 0.053).

The percentage, but not the number, of CD163 + macrophages in BAL showed a trend for correlation with sputum (Rs = 0.267, p = 0.053).