“showed a trend”

Conversely, the use of cryobiopsy showed a trend towards improved diagnostic yield (OR 1.88 (95% CI 0.94–3.66); p=0.064).

Cl-amidine treated females also showed a trend towards reduced neutrophil proportion out of CD45 + immune cells ( Fig. 3F , p = 0.064).

Compared with mFS 1, mFS 4 was associated with a markedly increased risk of unfavorable outcomes (OR = 25.05, 95%CI: 3.47–180.57, p = 0.001); mFS 3 showed a significant association (OR = 5.36, 95%CI: 1.02–28.14, p = 0.047), while mFS 2 showed a trend without reaching statistical significance (OR = 4.53, 95%CI: 0.91–22.45, p = 0.064).

Pairwise comparisons showed this to be due to GluN1 hypo female mice exploring more than WT female mice (WT 21.0 ± 3.3 s, GluN1 hypo 47.0 ± 7.2 s; P < 0.001), WT male mice (25.0 ± 2.4 s, P = 0.002) and showed a trend toward exploring more than GluN1 hypo male mice (31.5 ± 3.5 s, P = 0.064).

The correlation between Id1/Id3 co-expression and the PFS also showed a trend towards the statistical significance (R = −0.639, p = 0.064).

The main effect of sex showed a trend toward higher permeability in females ( F (1, 41) = 3.64, p = 0.064), with mean L p values of 1.11 × 10 −6 ± 1.24 × 10 −7 versus 8.05 × 10 −7 ± 1.08 × 10 −7 cm/cmH 2 O/s for females and males, respectively.

Conversely, older paternal age (>35 years at childbirth) showed a trend toward increased offspring risks, particularly for SSD (X 2 = 3.426, p = 0.064).

These findings were further validated across multiple independent cohorts: the Lauss 2017 CAR-T therapy cohort showed a trend toward improved PFS (P = 0.064), while the Cho 2020 (anti-PD-1/PD-L1, PFS, P = 0.021), IMvigor210 2018 (anti-PD-L1, OS, P = 0.0051), and Nathanson 2017 (anti-CTLA-4, OS, P = 0.04) cohorts consistently confirmed that high SLFN11 expression is associated with favorable prognosis in immunotherapy settings ( Figure 4B ).

control, p < 0.05), while O+B showed a trend of increased global H3K4me2 compared to the olanzapine-only group ( p = 0.064) ( Figure 4 A).

Survival analyses revealed that patients concurrently harboring PIK3R2 alterations showed a trend toward shorter progression-free survival (6 vs. 13 months, P=0.064) and significantly shorter overall survival (11 vs. 32 months, P=0.004) than did PIK3R2 -wild-type patients.

NS-398 showed a trend to reduce inflammation in TNBS colitis rats ( p = 0.064 vs.

Nonetheless, when considering patients with a lower severity of illness (APACHE II < 12), the occurrence of SSD showed a trend towards increased mortality ( p = 0.064).

Analysis of clonal heterogeneity in matched DX‐REL samples showed a trend towards reduced entropy levels in REL samples ( P = 0.064), with six of 10 patients showing lower entropy levels in the REL samples (Fig. 6D ), supporting previous data on DX samples of high levels of clonal heterogeneity as a favorable prognostic factor in HHD‐B‐ALL.

Quantitative evaluation of CD68 showed a trend for increased CD68 ( P = 0.064 Mann–Whitney) in these lesions compared with normal cortex, a decline in MAP2 ( P = 0.07, Mann–Whitney) but no significant alteration in GFAP with days post‐injury in this small group (Figure 4 ).

None of the other variants had compelling evidence of replication in our Stage2 data ( Table 3 ), although rs7861480 near IFNE showed a trend in the same direction ( P = 0.064).

Yet, a longer time interval from the last ON showed a trend of less total HRF ( β = −0.05 ± 0.03 [ p = 0.064]).

The tandem ASCT cohort showed a trend of better 3‐year probability of PFS (58.1% vs. 64.7%, p = 0.064) compared with the single ASCT cohort.

The clinical prediction model also showed a trend to be improved by addition of cTnT separately, yielding a continuous net reclassification index improvement of 8.0% (95% CI, –2.3% to 11.9%; P =0.064), and an integrated discrimination index of +0.001 (95% CI, 0.000–0.002; P =0.001).

When AUROC values of the SOFA score, lactate, and SAH were tested for differences between patient groups with low and high BOD, the mean and NAS of SAH showed a trend toward statistical significance ( p = 0.064 and 0.056, Table 3 ).

In the model 2 of multivariable analysis, considering the detailed metastatic sites instead of number of metastatic sites, we found exon 21 (HR [95% CI]: 1.56 [1.04–2.36], p = 0.0336), pleural metastasis/effusion (HR [95% CI]: 1.77 [1.17–2.69], p = 0.0075), and bone metastasis (HR [95% CI]: 1.69 [1.13–2.55], p = 0.0116) were independent risk factors for poorer PFS, while dose reduction showed a trend toward better PFS (HR [95% CI]: 0.59 [0.34–1.03], p = 0.0640).

Single correlations between WSC and CP showed a trend ( p = 0.064) to vary among cultivars: −0.45 for the 4nHSEU cultivar and from −0.75 to −0.78 for the remaining cultivars.

Frequent eating of a diet containing wheat or barley (AOR = 2.3, 95% CI: 0.9–5.4) showed a trend to be a risk factor for first-line ART failure (p-value = 0.064).

Between group differences assessed with Mann–Whitney U -Test showed that compared to the control group, total AβIR was significantly higher in AD ( p = 0.002), showed a trend with AD-VaD ( p = 0.064) and PSND ( p = 0.076) but not significantly different between PSD and VaD.

Compared to non-progressors, the patients who progressed to worse CKD stages showed a trend toward higher levels of urinary KIM-1 (P=0.064) and NGAL (P=0.065) at baseline; there was no difference in NAG levels.

Results Out of all measured steroids only concentrations of cortisol showed a trend to be associated with the symptom score at the baseline (r = −0.575, p = 0.064, n = 11); adding to the data concentrations observed in two unaffected siblings improved the association (r = −0.621, p = 0.024).