Complementary NAD⁺ replacement strategies fail to functionally protect dystrophin-deficient muscle.

Consistent with this model, we observed a significant increase in ion counts for glucose-6-phosphate (G6P), dihydroxyacetone phosphate (DHAP), ribose-5-phosphate, and a positive trend in sedoheptulose-7-phosphate (S7P) in MDX muscle (Fig. 1 e, g).

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Mice treated with GSK978A, but not NR, showed a trend toward protection from this effect, though it could not be attributed to specific NAD elevation in either limb (Fig. 3 e).

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